ACTC1 (E99K mutation) Rabbit Polyclonal Antibody AP54763PU-N from OriGene Technologies | Biocompare.com

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ACTC1 (E99K mutation) Rabbit Polyclonal Antibody

The ACTC1 (E99K mutation) Rabbit Polyclonal Antibody from OriGene Technologies is a Rabbit Polyclonal antibody to ACTC1, and actin alpha cardiac muscle 1. This antibody recognizes Human, Mouse, and Rat antigen. The ACTC1 (E99K mutation) Rabbit Polyclonal Antibody has been validated for the following applications: EIA, Immunoassay, ELISA, Immunohistochemistry, and Western Blot.

Description

ACTC1 (E99K mutation) rabbit polyclonal antibody, Purified

Product Specs
ItemACTC1 (E99K mutation) Rabbit Polyclonal Antibody
CompanyOriGene Technologies
Price Pricing Info Supplier Page View Company Product Page
Catalog NumberAP54763PU-N
Quantity100 µg
ApplicationsELISA, IHC, WB
Conjugate/TagUnconjugated
PurityAffinity chromatography on Protein A
ReactivityHuman, Mouse, Rat
HostRabbit
Antibody TypePrimary Antibodies
ImmunogenSynthetic peptide from human ACTC1 protein containing E99K mutation
Brand/TrademarkAcris Antibodies GmbH
NCBI Full Gene Nameactin alpha cardiac muscle 1
NCBI Gene AliasesLVNC4, CMD1R, CMH11, ACTC, ASD5
ClonalityPolyclonal
TypePrimary Antibodies
FormatTris 0,1M, glycine 0,1M, sucrose 2%
State: Purified
State: Lyophilized purified powder
Concentrationlot specific
TargetACTC1

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OriGene Technologies

OriGene Technologies

9620 Medical Center Drive # 200

Rockville, Maryland 20850

United States

Phone: 1-888-267-4436 (U.S. only) / 301-340-3188
301-340-9254

Company Profile

Website: http://www.origene.com

(1) Isogenic Pairs of hiPSC-CMs with Hypertrophic Cardiomyopathy/LVNC-Associated ACTC1 E99K Mutation Unveil Differential Functional DeficitsStem cell reportsNovember 13, 2018Smith JGW, Owen T, Bhagwan JR, Mosqueira D, Scott E, Mannhardt I, Patel A, Barriales-Villa R, Monserrat L, Hansen A, et al.

(2) Molecular mechanism of the E99K mutation in cardiac actin (ACTC Gene) that causes apical hypertrophy in man and mouseThe Journal of biological chemistryAugust 5, 2011Song W, Dyer E, Stuckey DJ, Copeland O, Leung MC, Bayliss C, Messer A, Wilkinson R, Tremoleda JL, Schneider MD, et al.

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